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Main Library
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The genetic contribution to non-syndromic human obesity.
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- Author(s): Walley, Andrew J.1 ; Asher, Julian E.1; Froguel, Philippe2
- Source:
Nature Reviews Genetics. Jul2009, Vol. 10 Issue 7, p431-442. 12p. 1 Diagram, 4 Charts, 1 Graph.- Subject Terms:
- Source:
- Additional Information
- Abstract: The last few years have seen major advances in common non-syndromic obesity research, much of it the result of genetic studies. This Review outlines the competing hypotheses about the mechanisms underlying the genetic and physiological basis of obesity, and then examines the recent explosion of genetic association studies that have yielded insights into obesity, both at the candidate gene level and the genome-wide level. With obesity genetics now entering the post-genome-wide association scan era, the obvious question is how to improve the results obtained so far using single nucleotide polymorphism markers and how to move successfully into the other areas of genomic variation that may be associated with common obesity. [ABSTRACT FROM AUTHOR]
- Abstract: Copyright of Nature Reviews Genetics is the property of Springer Nature and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)
- Abstract:
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